<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Hoyle, Brian</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Barkhof, Frederik</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Postrelapse MS Treatment with Alemtuzumab Better than SC IFN-β-1a</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2014</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2014-06-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">14-15</style></pages><abstract><style  face="normal" font="default" size="100%">Subgroup analyses, entitled An Extension Protocol for Multiple Sclerosis Patients Who Participated in Genzyme-Sponsored Studies of Alemtuzumab [NCT00930553; Barkhof F et al. ACTRIMS/ECTRIMS 2014 (poster P075)], of an ongoing, open-label extension of the Comparison of Alemtuzumab and Rebif Efficacy in Multiple Sclerosis, Study Two [CARE-MS II; Coles AJ et al. Lancet. 2012], have confirmed the superiority of treatment of MS using the anti-CD52 humanized monoclonal antibody, alemtuzumab, over subcutaneous interferon beta-1a (SC IFN-β-1a) in magnetic resonance imaging (MRI) outcomes.</style></abstract><number><style face="normal" font="default" size="100%">29</style></number><volume><style face="normal" font="default" size="100%">14</style></volume></record></records></xml>