<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Kuznar, Wayne</style></author></authors><secondary-authors></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Investigational Selective JAK1 Inhibitor Improves Measures of Disease Activity in RA</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2013</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2013-08-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">18-18</style></pages><abstract><style  face="normal" font="default" size="100%">An investigational selective inhibitor of Janus kinase 1 (JAK1) was found to improve clinical endpoints with a favorable safety profile in a Phase 2a dose-ranging study of patients with rheumatoid arthritis (RA). This article discusses the results of a 4-week double-blind, placebo-controlled study of GLPG0634, an orally-available, selective inhibitor of JAK1, in patients with active RA [Vanhoutte FP et al. EULAR 2013 (poster 0229)].</style></abstract><number><style face="normal" font="default" size="100%">8</style></number><volume><style face="normal" font="default" size="100%">13</style></volume></record></records></xml>