PT  - JOURNAL ARTICLE
AU  - Buckley, Rita
ED  - Kurosawa, Shinichiro
TI  - Complement Inactive in E. coli Shiga Toxin-Producing Diseases
DP  - 2013 Sep 01
TA  - MD Conference Express
PG  - 16--17
VI  - 13
IP  - 13
4099  - http://mdc.sagepub.com/content/13/13/16.short
4100  - http://mdc.sagepub.com/content/13/13/16.full
AB  - In a nonhuman primate model of hemolytic uremic syndrome, complement was not activated despite clear microvascular thrombosis and cellular injury. Complement is an important immune defense mechanism. This article presents outcomes from a study on the role of complement in HUS and thrombotic microangiography induced by Escherichia coli (E. coli) Shiga toxins.