<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Vinall, Phil</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Shah, Shreepal</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">The Precocious “AGE'ing” Effect of Type 1 Diabetes in Children</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2012</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2012-08-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">10-10</style></pages><abstract><style  face="normal" font="default" size="100%">Type 1 diabetes mellitus increases the formation of long-lived complexes, known as advanced glycation endproducts (AGEs), which can contribute to the development of complications in patients with diabetes [Genuth S et al. Diabetes 2005]. AGEs accumulate naturally in tissues over time, and tissue burden is a function of chronological age. This article presents results of the “The Precocious “AGE—ing” Effect of Type 1 Diabetes in Children” study, which examined the relationship between chronological age and type 1 diabetes on skin AGEs in children.</style></abstract><number><style face="normal" font="default" size="100%">10</style></number><volume><style face="normal" font="default" size="100%">12</style></volume></record></records></xml>