<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Buckley, Rita</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Kirino, Yohei</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Genome-Wide Analysis of Imputed Genotypes Identifies CCR1/CCR3 as Novel Risk Locus in Behçet Disease</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2011</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2011-12-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">17-18</style></pages><abstract><style  face="normal" font="default" size="100%">Behçet disease (BD) is a genetically complex condition that is characterized by recurrent inflammatory attacks that affect the orogenital mucosa, eyes, and skin. Genomewide association studies have linked the IL10 and IL23R-IL12RB2 loci to BD susceptibility [Remmers EF et al. Nat Genet 2010; Mizuki N et al. Nat Genet 2010].</style></abstract><number><style face="normal" font="default" size="100%">13</style></number><volume><style face="normal" font="default" size="100%">11</style></volume></record></records></xml>