<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Nichols, Emma Hitt</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Vaisse, Christian</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">New Members of the Leptin–Melanocortin Axis Linked to Pediatric Obesity</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2014</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2014-10-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">26-27</style></pages><abstract><style  face="normal" font="default" size="100%">Proper function of the leptin–melanocortin axis is important in regulating food intake and maintaining energy homeostasis. Therefore, mutations that affect the leptin–melanocortin axis result in several known syndromes that result in an obesity phenotype. Recent research has further identified important components of this axis that may result in childhood-onset obesity as discussed in this article.</style></abstract><number><style face="normal" font="default" size="100%">20</style></number><volume><style face="normal" font="default" size="100%">14</style></volume></record></records></xml>