<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Mosley, Mary</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Macdonald, R. Loch</style></author><author><style face="normal" font="default" size="100%">Provencio, Jose Javier</style></author><author><style face="normal" font="default" size="100%">Simard, J. Marc</style></author><author><style face="normal" font="default" size="100%">James, Robert F.</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Cognitive Dysfunction and Neuroinflammation After an aSAH</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2015</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2015-03-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">17-19</style></pages><abstract><style  face="normal" font="default" size="100%">Cognitive dysfunction occurs after an aneurysmal subarachnoid hemorrhage, affecting psychosocial behavior, activities of daily living, and return to work. The mechanisms for the cognitive deficits are not understood, but neuroinflammation appears to be one process. New therapies to target neuroinflammation are being studied.</style></abstract><number><style face="normal" font="default" size="100%">2</style></number><volume><style face="normal" font="default" size="100%">15</style></volume></record></records></xml>