<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Vinall, Maria</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Bourguignon, Jean-Pierre</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Neonatal Exposure to BPA Affects Puberty Onset in Rats</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2014</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2014-10-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">9-10</style></pages><abstract><style  face="normal" font="default" size="100%">Bisphenol A (BPA) is an industrial chemical used to make polycarbonate—a hard, clear plastic used in many consumer products. Endocrine-disrupting effects of low BPA doses in the microgram range are a matter of controversy. However, this article reports that an injection of a very low dose (25 ng/kg/day) of BPA into neonatal rats delayed puberty, slowed down gonadotropin-releasing hormone secretion, and changed hypothalamic RNA expression.</style></abstract><number><style face="normal" font="default" size="100%">20</style></number><volume><style face="normal" font="default" size="100%">14</style></volume></record></records></xml>