<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Mosely, Mary</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Eikelis, Nina</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Effects of Renal Denervation on Endothelial Function, and Inflammatory and Metabolic Markers</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2013</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2013-07-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">8-9</style></pages><abstract><style  face="normal" font="default" size="100%">It has been shown that renal denervation (RDN; also called renal nerve ablation) can lower blood pressure (BP) in patients with resistant hypertension and that this effect can be maintained to more than 24 months [Symplicity HTN-1 Investigators. Hypertens 2011]. However, there are few data about the effect of RDN on organs and the sympathetic nervous system. This article presents data from a study to determine whether RDN has an effect on inflammation and endothelial function in patients with resistant hypertension.</style></abstract><number><style face="normal" font="default" size="100%">9</style></number><volume><style face="normal" font="default" size="100%">13</style></volume></record></records></xml>