<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Buckley, Rita</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Watson, Steve P.</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Phenotyping and Genotyping of Platelet Disorders</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2013</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2013-08-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">29-30</style></pages><abstract><style  face="normal" font="default" size="100%">Investigation of patients with mild bleeding disorders might provide novel information on the regulation and role of platelet proteins. It might even identify new targets for prevention of thrombosis. However, gene mutations require phenotypic support to assign causation, according to findings from the observational study, Genotyping and Platelet Phenotyping [GAPP; ISRCTN77951167; UKCRN ID 9858]. This article presents results to date from the study.</style></abstract><number><style face="normal" font="default" size="100%">13</style></number><volume><style face="normal" font="default" size="100%">13</style></volume></record></records></xml>