Summary
In a nonhuman primate model of hemolytic uremic syndrome, complement was not activated despite clear microvascular thrombosis and cellular injury. Complement is an important immune defense mechanism. This article presents outcomes from a study on the role of complement in HUS and thrombotic microangiography induced by Escherichia coli (E. coli) Shiga toxins.
- Hematology Clinical Trials
- Anemias
- Bacterial Infections
- Hematology Clinical Trials
- Hematology
- Anemias
- Bacterial Infections
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