<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Rizzo, Toni</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Kim, Jae Bum</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Role of iNKT Cells in Adipose Tissue Inflammation and Insulin Resistance</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2013</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2013-11-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">10-10</style></pages><abstract><style  face="normal" font="default" size="100%">This article, presents results of experiments which suggest that invariant natural killer T (iNKT) cells decrease the adipose tissue inflammation. A study using transgenic mice fed a high-fat diet reported that infiltration of human resistin-expressing macrophages into adipose tissue is a key mechanism for development of accelerated adipose tissue inflammation [Qatanani M et al. Clin Invest 2009]. Resistin interacts with CAP1 on macrophages, stimulating the secretion of inflammatory cytokines that interferes with the normal insulin signaling pathway, demonstrating that adipose tissue inflammation may mediate insulin resistance.</style></abstract><number><style face="normal" font="default" size="100%">16</style></number><volume><style face="normal" font="default" size="100%">13</style></volume></record></records></xml>