<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Buckley, Rita</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Prabhakar, Nanduri R.</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Intermittent Hypoxia — A Sleep Apnea Model</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2011</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2011-11-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">23-24</style></pages><abstract><style  face="normal" font="default" size="100%">From 1986 to 2002, sleep apnea affected &gt;100 million individuals worldwide [WHO; Chronic Respiratory Diseases]. Apneas lead to periodic desaturation of O2 in arterial blood. In severe cases, O2 saturation drops to nearly 50%, and the frequency of apneas can reach as many as 60 to 90 episodes per hour. This article discusses the effects of intermittent hypoxia on glucose homeostasis.</style></abstract><number><style face="normal" font="default" size="100%">11</style></number><volume><style face="normal" font="default" size="100%">11</style></volume></record></records></xml>