<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Gordon, Debra</style></author></authors><secondary-authors></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">The Future of Antidiabetic Drug Development: GPR40-Selective Agonist TAK-875</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2010</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2010-11-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">35-36</style></pages><abstract><style  face="normal" font="default" size="100%">GPR40 is a G-protein-coupled receptor that is highly expressed in pancreatic β-cells and is involved in free fatty acid-induced insulin secretion. TAK-875 is a GPR40-selective agonist that improves glucose control in type 2 diabetic animal models by stimulating glucose-dependent insulin secretion.</style></abstract><number><style face="normal" font="default" size="100%">9</style></number><volume><style face="normal" font="default" size="100%">10</style></volume></record></records></xml>