<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Vinall, Maria</style></author></authors><secondary-authors><author><style face="normal" font="default" size="100%">Hills, Nancy K.</style></author></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Issues in Pediatric Stroke</style></title><secondary-title><style face="normal" font="default" size="100%">MD Conference Express</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2009</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2009-03-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">27-28</style></pages><abstract><style  face="normal" font="default" size="100%">Cerebral arteriopathy is the strongest predictor of recurrent arterial ischemic stroke (AIS) in childhood [Fullerton HJ et al. Pediatrics 2007]. This article discusses results from the International Pediatric Stroke Study [IPSS], which suggest a role for infection in the pathogenesis of cerebral arteriopathy. Also discussed are the results of a retrospective study that showed that circulating endothelial cells can be used to track cerebral arteriopathy-associated vascular injury and to differentiate progressive versus nonprogressive disease in children with AIS.</style></abstract><number><style face="normal" font="default" size="100%">1</style></number><volume><style face="normal" font="default" size="100%">9</style></volume></record></records></xml>